Peritonsiler abscess is an infection that started from the outer surface that extends into the inner layer of soft tissue. Exactly pathophysiology of PTA is unknown. However, several theories attempt to explain the mechanism of the PTA, and the theory was widely accepted that the occurrence of an abscess peritonsiler tonsil inflammation spreading from there. Abscess formed between tonsila Palatina and the capsule, usually in the superior region. It is then believed that the abscesses originated from an acute episode include tonsilits growing soft tissue around the area (Mehta, 2007). Other Mecanisme happened is the emergence of necrosis and pus in the capsular area, which then clog the Weber glands, minor salivary glands found in the living and working peritonsiler help clean up debris from the tonsils, resulting in secretions and forming abscesses (Gosselin, 2008).
Superior and lateral regions tonsilaris fossa is loose connective tissue, the infiltration into the potential supurasi posters peritonsil occupy this area, so look swollen palate molle. In the early stage (stage infiltrates), in addition to swelling, the surface looks hiperemis. If the process continues, the area is more soft and yellowish color. Tonsils pushed to the middle, front and bottom, and swollen uvula pushed to the contralateral side. If the process continues, inflammation of the surrounding tissue will cause irritation of the m. Pterigoid internal, resulting in trismus. Abscesses may rupture spontaneously, may occur to the pulmonary aspiration (Fachruddin, 2002).
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